Max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. On the flip side, our capability to rescue SERT operate in KI midbrain synaptoneurosomes because of the inhibition of FAK indicates elevated FAK signaling downstream in the Pro32Pro33 mutant, as verified by increased pFAK localization in https://elizabethj420nyk3.wikipowell.com/user